Why do microglia from a disease mouse model show increased respiration parameters (basal, maximal, and ATP-linked OCR) in a Seahorse Mito Stress Test while a separate biochemical assay shows no differ
Why do microglia from a disease mouse model show increased respiration parameters (basal, maximal, and ATP-linked OCR) in a Seahorse Mito Stress Test while a separate biochemical assay shows no difference in ATP production?
Why do microglia from a disease mouse model show increased respiration parameters (basal, maximal, and ATP-linked OCR) in a Seahorse Mito Stress Test while a separate biochemical assay shows no difference in ATP production?
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